Preliminary experiments show conolidine may well inhibit distinct ion channels, lessening neuronal excitability and restricting discomfort indicators. This mechanism is especially suitable in neuropathic agony, where irregular signaling will cause persistent pain. In addition, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to pain notion. Computati... https://raymondp739neu4.wikibyby.com/user